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Cardiac arrhythmias are identified as an abnormal activation or beating of heart myocardium. The primary factors of arrhythmia, including myocardial injury, mechanical stress, neurohormonal activation, inflammation, and/or aging, result in cardiac remodeling, which is responsible for cardiac dysfunction and arrhythmogenesis. Dizziness, palpitations, fast heart beating, and feeling of weakness are the main symptoms of arrhythmia. In recent years, the incidence of arrhythmia has been rising rapidly worldwide. Therefore, the development of targeted treatment strategies for cardiac arrhythmia is necessary.
Creative Bioarray offers you cardiac arrhythmia model to help you understand the pathological mechanisms of arrhythmia and test the efficacy of novel drugs against it.
With extensive experience in the field of cardiac arrhythmia, we are confident to help you to overcome any upcoming challenges. Our experts are fully capable of customizing our protocols and assays to meet your specific needs. With our help, we wish to facilitate your research with high efficiency.
Figure. 1. Effects of pinocembrin on HR and MAP of I/R rats. T0 – 10 min before ischemia; T1 – 30 min after ischemia; T2 – 30 min after reperfusion; T3 – 60 min after reperfusion; T4 – 120 min re-perfusion. HR – heart rate; MAP – mean artery pressure. A – Sham group; B – model group; C – pinocembrin group.
Figure. 2. Effects of pinocembrin on I/R rat RPP and cardiac arrhythmia. T0 – 10 min before ischemia; T1 – 30 min after ischemia; T2 – 30 min after reperfusion; T3 – 60 min after re-perfusion; T4 – 120 min reperfusion. RPP – rate pressure product. A – sham group; B – model group; C – pinocembrin group.
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Zhang P , Xu J , Hu W , et al. Effects of Pinocembrin Pretreatment on Connexin 43 (Cx43) Protein Expression After Rat Myocardial Ischemia-Reperfusion and Cardiac Arrhythmia[J]. Medical Science Monitor International Medical Journal of Experimental & Clinical Research, 2018, 24:5008-5014.
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